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Conclusions This narrative review demonstrates that semaglutideโs oral adverse effects cannot yet be explained by direct salivary-gland data, but the convergence of three mechanistic domainsโalbumin-mediated continuous receptor stimulation, bias toward sustained Gs/cAMP signaling, and ฮฒ-arrestinโdependent desensitizationโprovides a coherent hypothesis for salivary hypofunction
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