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Unraveling a causal relationship between GLP-1 agonists and AP is intricate, particularly as patients with T2DM are already three times more predisposed to pancreatitis compared to their non-diabetic counterparts (Girman et al., 2010)
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This GLP-1R model was embedded in a 95 × 95 Å palmitoyl oleoyl phosphatidyl choline (POPC) bilayer and lipids located within 1 Å of the receptor were removed
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