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Altered glucose metabolism after bariatric surgery: What's GLP-1 got to do with it? - ScienceDirect

Third, the underlying mechanism of DPP4 D and Sita treatment effectively preserving the brain architecture and the integrity of neurological function in setting of AHS was identified mainly due to downregulations of the inflammatory, oxidative-stress and cell stress signaling

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For example, GLP-1R agonists were able to improve glucose metabolism function through the GLP-1R/SIRT1/GLUT4 pathway in an AD model (Wang Z