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In ARC POMC neurons, leptin activates α-MSH to agonize MC4R and MC3R, increases sympathetic nervous outflow to the kidney and the heart, and exacerbates hypertension [259]
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Platelet studies suggest that GLP-1 receptor activation can reduce platelet aggregation through nitric oxide/cyclic GMP-dependent mechanisms, providing a plausible anti-thrombotic component (43)
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