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Similar to our previous RNA-seq analysis, we demonstrate that treatment with GLP-1–MK-801 significantly regulates around 150 times the number of hypothalamic transcripts relative to the weight-matched semaglutide-treated group, emphasizing a mode of action for GLP-1–MK-801 that involves biological pathways independent of the GLP-1 receptor signalling pathway (Fig

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As a continuum and not as a dichotomous phenomenon, the levels of C-peptide in the PDM diagnosis should be considered in the context of this added complexity