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Hiroaki Matsunami, PhD, professor of molecular genetics and microbiology, neurobiology, and cell biology at the Duke University School of Medicine, explains that there are “tens of thousands” of different scent elements—some good, some bad, some open to interpretation—floating around us at all times

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Exendin-4 enhances the binding of heat shock factor 1 to the promoter of heat shock protein (HSP) genes through SIRT1-mediated deacetylation, which then increased the expression of molecular chaperones HSP70 and HSP40 to alleviate lipid-induced hepatic ER stress.78 ER oxidoreductase mediates the inhibitory effects of exendin-4 on ER stress, ameliorating hyperhomocysteinemia-induced endothelial dysfunction.79 ER protein 46, a new member of the thioredoxin family, highly expressed in pancreatic β-cells, may mediate GLP-1 regulation of ER stress and thus increase the protection of pancreatic β cells.80,81 These studies suggest the complexity of GLP-1 RA in regulating ER stress

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