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In addition to reducing plaque size and necrotic core area in Apoe −/− Irs2 +/− mice, lixisenatide was found to result in a greater proportion of M2-like anti-inflammatory (STAT3 + Arginase-1 + ) macrophages in aortic plaques, with fewer M1-like, pro-inflammatory (STAT1 + iNOS + ) macrophages [134]
